• Title of article

    Cell of Origin in AML: Susceptibility to MN1-Induced Transformation Is Regulated by the MEIS1/AbdB-like HOX Protein Complex

  • Author/Authors

    Heuser، نويسنده , , Michael and Yun، نويسنده , , Haiyang and Berg، نويسنده , , Tobias and Yung، نويسنده , , Eric and Argiropoulos، نويسنده , , Bob and Kuchenbauer، نويسنده , , Florian and Park، نويسنده , , Gyeongsin and Hamwi، نويسنده , , Iyas and Palmqvist، نويسنده , , Lars and Lai، نويسنده , , Courteney K. and Leung، نويسنده , , Malina and Lin، نويسنده , , Grace and Chaturvedi، نويسنده , , Anuhar and Thakur، نويسنده , , Basant Kumar and Iwasaki، نويسنده , , Masayuki and Bilenky، نويسنده , , Mikhail and Thiessen، نويسنده , , Nina and Robertson، نويسنده , , Gordon and Hirst، نويسنده , , Martin and Kent، نويسنده , , David and Wilson، نويسنده , , Nicola K. and Gِttgens، نويسنده , , Bertie and Eaves، نويسنده , , Connie and Cleary، نويسنده , , Michael L. and Marra، نويسنده , , Marco and Ganser، نويسنده , , Arnold and Humphries، نويسنده , , R. Keith، نويسنده ,

  • Issue Information
    روزنامه با شماره پیاپی سال 2011
  • Pages
    14
  • From page
    39
  • To page
    52
  • Abstract
    Summary ys defining susceptibility of normal cells to oncogenic transformation may be valuable therapeutic targets. We characterized the cell of origin and its critical pathways in MN1-induced leukemias. Common myeloid (CMP) but not granulocyte-macrophage progenitors (GMP) could be transformed by MN1. Complementation studies of CMP-signature genes in GMPs demonstrated that MN1-leukemogenicity required the MEIS1/AbdB-like HOX-protein complex. ChIP-sequencing identified common target genes of MN1 and MEIS1 and demonstrated identical binding sites for a large proportion of their chromatin targets. Transcriptional repression of MEIS1 targets in established MN1 leukemias demonstrated antileukemic activity. As MN1 relies on but cannot activate expression of MEIS1/AbdB-like HOX proteins, transcriptional activity of these genes determines cellular susceptibility to MN1-induced transformation and may represent a promising therapeutic target. lip
  • Journal title
    Cancer Cell
  • Serial Year
    2011
  • Journal title
    Cancer Cell
  • Record number

    1337558