Author/Authors :
Biagini، نويسنده , , Andrea and Iervasi، نويسنده , , Giorgio and Clerico، نويسنده , , Aldo and Berti، نويسنده , , Sergio and Pilo، نويسنده , , Alessandro and Vitek، نويسنده , , Frantisek and Rita، نويسنده , , Bonini and Bianchi، نويسنده , , Romano and Donate، نويسنده , , Luigi، نويسنده ,
Abstract :
In conclusion, our kinetic data indicate that patients with complex ventricular arrhythmias show a peculiar alteration of thyroid hormone metabolism, characterized by increased T4 to T3 conversion and reduced T4 production, and resulting in a relatively increased availability of T3 at the cellular level. Therefore, our data support the hypothesis that an altered peripheral metabolism of thyroid hormones may play a role in the pathogenesis of complex ventricular arrhythmias, even if further studies are required to confirm this hypothesis and to supply information for their interpretation.
