Author/Authors :
Anker، نويسنده , , Stefan D. and Egerer، نويسنده , , Karl R. and Volk، نويسنده , , Hans-Dieter and Kox، نويسنده , , Wolfgang J. and Poole-Wilson، نويسنده , , Philip A. and Coats، نويسنده , , Andrew J.S.، نويسنده ,
Abstract :
We hypothesized that a chronic endotoxin challenge may cause immune activation in CHF. Patients with high soluble CD14 levels have markedly increased levels of TNF-α, soluble TNF receptors 1 and 2, and intracellular adhesion molecule-1, supporting this hypothesis.
