Title of article :
Ultra-early weaning in piglets results in low serum IgA concentration and IL17 mRNA expression
Author/Authors :
Levast، نويسنده , , Benoît and de Monte، نويسنده , , Michèle and Chevaleyre، نويسنده , , Claire and Melo، نويسنده , , Sandrine and Berri، نويسنده , , Mustapha and Mangin، نويسنده , , Françoise and Zanello، نويسنده , , Galliano and Lantier، نويسنده , , Isabelle and Salmon، نويسنده , , Henri and Meurens، نويسنده , , François، نويسنده ,
Issue Information :
سالنامه با شماره پیاپی سال 2010
Pages :
8
From page :
261
To page :
268
Abstract :
In pigs raised for meat production, weaning is a critical period because of related physiological perturbations and negative consequences on performance. Previous studies have shown that early weaning could either impair development of mucosal barrier function or boost intestinal immunologic parameters. In order to obtain further knowledge about the impact of ultra-early weaning on the porcine immune system development, three groups of piglets were weaned at different ages and compared to the unweaned control group. Lower IgA concentrations in ultra-early and early weaned piglets than in other piglets were identified in serum. In the mesenteric lymph node (MLN), significant differences in the mRNA expression of IL17a, TGF beta and FOXP3 were found between specific groups. Indeed, IL17a mRNA was mainly detected in ultra-early weaned piglets while FOXP3 and TGF beta mRNA were associated to both ultra-early weaned and suckling piglets. Reduced serum IgA concentration and MLN induction of a Th17 cytokine in ultra-early weaned piglets could be related to alterations of the mucosal barrier functions consecutive to the milk deprivation. All together, our findings suggest a crucial role for endogenous milk factors onto the onset of IgA synthesis.
Keywords :
weaning , IgA , cytokine , Mesenteric lymph node , piglets
Journal title :
Veterinary Immunology and Immunopathology
Serial Year :
2010
Journal title :
Veterinary Immunology and Immunopathology
Record number :
2165635
Link To Document :
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