Author/Authors :
Khlifi, Latifa University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia , Chabchoub, Elyes University of Sousse - Faculty of Medicine - Department of Genetics, Tunisia , Letaief, Afef University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia , Ben Othmen, Leila University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia , Smach, Mohamed Ali University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia , Charfeddine, Bassem University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia , Zemni, Ramzi University of Sousse - Faculty of Medicine - Department of Genetics, Tunisia , Khalifa, Limem University of Sousse - Faculty of Medicine - Department of Biochemistry, Tunisia
Abstract :
Coronary atherosclerosis is a chronic disease that can be considered as a form of chronic inflammation with stable and unstable phases corresponding to activated inflammation in the vascular wall (1). Evidence has been shown that cytokines, which are immunoregulatory proteins, play a complex role in the development of atherosclerotic lesions (1).
