Author`s Reply:Aggregation of lipoprotein(a) to apolipoprotein A-I and coronary artery problem

We appreciated to learn the comment by Wiwanitkit to our prospective population-based study published in September issue of The Anatolian Journal of Cardiology 2013; 13: 543-51 (1) indicating that aggregation of lipoprotein (Lp) (a) in a proinflammatory setting to apolipoprotein (apo) A-I, the major protein constituent of HDL particles, may lead to impairment of the antioxidant and atheroprotective functions of apoA-I, which may ultimately become diabetogenic or atherogenic, a process representing HDL dysfunction and autoimmune activation.