Letter to the Editor:Diastolic dysfunction and left atrial appendages: Time to phenotype the process of fibrosis

We read with interest the article by Demirçelik et al. (1) published in Anadolu Kardiyol Derg 2014 Jan 10. We believe that any effort aimed to elucidate the pathophysiological mechanisms of left atrial appendage (LAA) thrombus formation is valuable since these thrombi are the source of most embolic strokes in patients with nonvalvular atrial fibrillation (AF) and, at this regard, any decrease in filling and emptying velocities of LAA may affect blood coagulability.