Intraocular inflammatory reaction without focal necrotizing retinochoroiditi in patient with acquired ytemic toxoplamoi Original Reearch Article

PURPOE: To decribe the occurrence of intraocular inflammatory reaction a the ole ophthalmic manifetation of acquired ytemic toxoplamoi. METHOD: Review of medical record for 10 patient with uveiti and evidence of recent Toxoplama gondii infection. REULT: Patient age ranged from 3 to 51 year. Ocular ymptom were preent in each of eight adult patient. Inflammation wa unilateral in nine patient; it manifeted a vitreou humor cell and haze (10 patient), anterior chamber cell (even patient), and retinal vaculiti (even patient). No patient had necrotizing retinochoroiditi upon initial examination. Inflammation reolved in each of nine patient who had follow-up examination. Foci of retiniti or inactive retinochoroidal car were een in four of thee nine patient during follow-up examination, at interval of 2.0 week to 2.5 year after initial examination. CONCLUION: Retinal vaculiti and aociated inflammatory reaction may be the only ophthalmic diorder during the early tage of a newly acquired T. gondii infection. Later development of retiniti or car conitent with toxoplamic retinochoroiditi in the ame eye ugget that the initial, iolated inflammation may be caued by the preence of paraite in retinal tiue. Thee cae may have implication for undertanding the original ource of retinal infection in patient who have recurrent toxoplamic retinochoroiditi and for treatment of newly acquired T gondii infection.