• Title of article

    Fetal pyelectasis: Is it always “physiologic”?

  • Author/Authors

    Abdallah M. Adra، نويسنده , , Andres A. Mejides، نويسنده , , Maha S. Dennaoui، نويسنده , , Samir N. Beydoun، نويسنده ,

  • Issue Information
    روزنامه با شماره پیاپی سال 1996
  • Pages
    4
  • From page
    1263
  • To page
    1266
  • Abstract
    OBJECTIVE: Our purpose was to determine the degree of fetal pyelectasis predictive of neonatal renal pathologic processes. STUDY DESIGN: Eighty-four cases of pyelectasis were identified during the study period (1989 through 1993). Fetuses with an anteroposterior diameter of the renal pelvis ≥4 mm before 33 weeks or ≥7 mm after 33 weeks without caliectasis were included. Fetuses with an anteroposterior diameter of>10 mm were excluded. Postnatal evaluation included renal sonogram, voiding cystourethrogram, and renal flow and function studies. RESULTS: Sixteen cases were excluded because of incomplete postnatal workup. Renal pathologic processes were found in 30 of 68 (44%) at birth: the most common were ureteropelvic junction obstruction (37%) and vesicoureteral reflux (33%). Only four cases required surgical intervention (13%), and the remaining 87% were managed conservatively. A normal urinary tract was found in 25% of the infants and the remaining 21 of 68 (31%) had regression of pyelectasis before birth. Fetuses with a dilated anteroposterior diameter ≥8 mm after 28 weeksʹ gestation were found to have renal pathologic features in two of three of the cases, with sensitivity, specificity, and positive and negative predictive values of 87%, 41%, 66.7% and 70%, respectively. CONCLUSION: Fetuses with an anteroposterior diameter of the renal pelvis ≥8 mm after 28 weeksʹ gestation require appropriate urologic evaluation after birth.
  • Keywords
    renal pathology , Prenatal ultrasonography , pyelectasis
  • Journal title
    American Journal of Obstetrics and Gynecology
  • Serial Year
    1996
  • Journal title
    American Journal of Obstetrics and Gynecology
  • Record number

    639191