Title of article :
Human-like rodent amyloid-β-peptide determines Alzheimer pathology in aged wild-type Octodon degu
Author/Authors :
Nibaldo C. Inestrosaa، نويسنده , , ?، نويسنده , , Ariel E. Reyesa، نويسنده , , Marcelo A. Chac´ona، نويسنده , , Waldo Cerpaa، نويسنده , , Aldo Villal´onb، نويسنده , , Juan Montielb، نويسنده , , Genevieve Merabachvilia، نويسنده , , Rebeca Aldunatea، نويسنده , , Francisco Bozinovicc، نويسنده , , Francisco Aboitiz، نويسنده ,
Issue Information :
روزنامه با شماره پیاپی سال 2005
Pages :
6
From page :
1023
To page :
1028
Abstract :
It is generally accepted that human Alzheimerʹs disease (AD) neuropathology markers are completely absent in rodent brains. We report here that an aged wild-type South American rodent, Octodon degu, expresses neuronal β-amyloid precursor protein (β-APP695) displaying both intracellular and extracellular deposits of amyloid-β-peptide (Aβ), intracellular accumulations of tau-protein and ubiquitin, a strong astrocytic response and acetylcholinesterase (AChE)-rich pyramidal neurons. The high amino acid homology (97.5%) between deguAβ and humanAβ sequences is probably a major factor in the appearance of AD markers in this aged rodent. Our results indicate that aged O. degu constitutes the first wild-type rodent model for neurodegenerative processes associated to AD.
Keywords :
neuropathology , Amyloid- -peptide , APP , Octodon degu , Alzheimer’s Disease , Alzheimer model
Journal title :
Neurobiology of Aging
Serial Year :
2005
Journal title :
Neurobiology of Aging
Record number :
820659
Link To Document :
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