• DocumentCode
    2505497
  • Title

    Effect of Overfeeding on the Transcriptional Expression of aP2 Gene in Landes Goose Liver

  • Author

    Su, Shengyan ; Li, Xuebin ; Li, Qifa ; Xie, Zhuang

  • Author_Institution
    Coll. of Animal Sci. & Technol., Nanjing Agric. Univ., Nanjing, China
  • fYear
    2009
  • fDate
    11-13 June 2009
  • Firstpage
    1
  • Lastpage
    3
  • Abstract
    The purpose of this experiment was to determine the effects of overfeeding on hepatic fat deposition at lipogenic genes expression. Overfeeding had effects on the body weight, liver weight, abdominal adipose weight and liver/body weight compared to control. In contrast, the dietary treatment had markedly difference in ALT, TG and H-cholesterol. In liver, the expression of aP2 mRNA was higher in geese overfeeding than that of control group (P < 0.01). The correlation coefficient of aP2 transcriptional level in liver and liver/body weight was 0.598(P<0.05). There were significant correlations between Che and liver expression of aP2. The genetic regulatory pathway may contribute directly or indirectly to the forming of landes geese fore gras mainly on the phase of preadipocyte differentiation into adipocytes. However, factors influenced the forming of landes geese fat livers by consumption of a high carbohydrate diet were more.
  • Keywords
    cellular biophysics; genetics; liver; macromolecules; organic compounds; patient treatment; ALT; H-cholesterol; aP2 gene transcriptional expression; abdominal adipose weight; adipocytes; dietary treatment; hepatic fat deposition; landes goose liver; lipogenic genes expression; mRNA; overfeeding effect; preadipocyte differentiation; Abdomen; Animals; DNA; Educational institutions; Genetic expression; Liver; Proteins; RNA; Temperature; Weight control;
  • fLanguage
    English
  • Publisher
    ieee
  • Conference_Titel
    Bioinformatics and Biomedical Engineering , 2009. ICBBE 2009. 3rd International Conference on
  • Conference_Location
    Beijing
  • Print_ISBN
    978-1-4244-2901-1
  • Electronic_ISBN
    978-1-4244-2902-8
  • Type

    conf

  • DOI
    10.1109/ICBBE.2009.5162706
  • Filename
    5162706