DocumentCode :
513608
Title :
Childhood leukemia and CNS sanctuary therapy
Author :
Yadava, Ram L.
Author_Institution :
Sch. of Electr. Sci., Vellore Inst. of Technol., Vellore, India
fYear :
2006
fDate :
23-24 Feb. 2006
Firstpage :
443
Lastpage :
444
Abstract :
Cancer develops when cells in a part of the body begin to grow out of control. Normal body cells grow, divide, and die in an orderly fashion. During the early years of a person´s life, normal cells divide more rapidly until the person becomes an adult. After that, cells in most parts of the body divide only to replace worn-out or dying cells and to repair injuries. Because cancer cells continue to grow and divide, they are different from normal cells. Instead of dying, they outlive normal cells and continue to form new abnormal cells. Cancer cells develop because of damage to DNA. This substance is in every cell and directs all its activities. Most of the time when DNA becomes damaged the body is able to repair it. In cancer cells, the damaged DNA is not repaired. People can inherit damaged DNA, which accounts for inherited cancers. Cancer usually forms as tumors, some cancers, like leukemia, do not form tumors. Instead, these cancer cells involve the blood and blood-forming organs and circulate through other tissues where they grow.
Keywords :
DNA; blood; bone; cancer; cellular biophysics; diseases; molecular biophysics; neurophysiology; radiation therapy; CNS sanctuary therapy; blood; blood-forming organs; cancer cells; childhood leukemia; damaged DNA; external beam radiation therapy; high-energy X-ray machine; tissues; tumors; Biomedical applications of radiation; Blood; Cancer; Cells (biology); DNA; Electromagnetic radiation; Linear accelerators; Medical treatment; Neoplasms; Nervous system;
fLanguage :
English
Publisher :
ieee
Conference_Titel :
ElectroMagnetic Interference and Compatibility (INCEMIC), 2006 Proceedings of the 9th International Conference on
Conference_Location :
Bangalore
Print_ISBN :
978-1-4244-5203-3
Type :
conf
Filename :
5419746
Link To Document :
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