بررسي رابطه هليكوباكترپيلوي و ضايعات دهان (آفت ، ليكن پالان ، زبان جغرافيايي و سوزش دهان )

Evaluation of Helicobacter Pylori Influence on Oral Lesion (Aphthous Ulcer. Oral Lichen Planus, Bearing Mouth Syndrome, and Geographic Tongue between the Patients

Itʹs a long time that some of the oral lesions with unknown etiologic factors are the targets of many researches, in oral disease diagnosis and different theories were examined. Also, the role of Helicobacter pylori (H.p) as the major etiologic factor in gastrodeudenal disease is undoutable. The oral cavity as the first part of GIT (Castro Intestinal Track) and the most important way in HP transmission (oral - oral, oral - fecal) is a critical area. Also, "HP" infection kike some oral lesions such as Aphthous ulcer, lichen planus, Bearing mouth syndrome and Geographic tongue is a recurrent disorder. The goal of this research was "evaluation of Helicobacter pylori influence on oral lesions (Aphthous ulcer (RAS), oral lichen planus (LP), Bearing mouth syndrome (BMS), Geographic tongue (GT). In this research " 26 " patients with oral lesions were selected (7 RAS, 7 LP, 6 BMS and 6 GT Patients). Beside this case group, "35" healthy patients with no oral lesion were selected as a control group. Two different samples were prepared from each case. (From the depth of the lesion and sub gingival plaque) and one sample was got from sub gingival plague in control group. Then we transferred the samples into specific cultures, which were: in microaerophilic area, 37 ° C, 5 - %C02 and sufficient humidity and after 3 to 5 days we observed the plates and applied the supplemental tests. (ureas test, catalase test proxidase test). The results showed that among "26" patients, only 3 samples were HP positive (12 %) and in " 25 " -patients in control group, we had no HP (+). (0 %) We considered some other factors, such as, stress, smoking, trauma, food allergy, medicine usage, systemic history, familial history of oral lesions and herpetic ulcers history. The results showed that these factors had no relation with HP in oral diseases.