DocumentCode
3324813
Title
Notice of Retraction
Relationship among MBL Gene Haplotypes, Genotype and Plasma Levels and Ischemic Cerebrovascular Disease
Author
Cheng Jianjun ; Cai Nianguang ; Jia Tianjun ; Nian Xiaofeng ; Kang Shaoping ; Yang Xinling ; Li Jing
Author_Institution
Coll. of Lab. Sci., Hebei North Univ., Zhangjiakou, China
fYear
2011
fDate
10-12 May 2011
Firstpage
1
Lastpage
4
Abstract
Notice of Retraction
After careful and considered review of the content of this paper by a duly constituted expert committee, this paper has been found to be in violation of IEEE´s Publication Principles.
We hereby retract the content of this paper. Reasonable effort should be made to remove all past references to this paper.
The presenting author of this paper has the option to appeal this decision by contacting TPII@ieee.org.
Objective: To detect the distribution of ICVD (Ischemic Cerebrovascular Disease) patients and healthy controls MBL haplotype and genotype frequencies, MBL plasma levels. Analyze the relevance between these factors´ change and ICVD and try to explain the possible pathogenesis of ICVD. Methods: MBL haplotypes were detected by SSP-PCR, Plasma MBL levels were detected by ELISA assay. Results: ICVD patients and healthy MBL haplotype and genotype proportions were different (p <;0.05); ICVD plasma MBL levels of patients (3372.18μg / L) was significantly higher (P <;0.01) than in healthy people (2065.29μg / L). Conclusion: MBL may be involved in the development process of ICVD.
After careful and considered review of the content of this paper by a duly constituted expert committee, this paper has been found to be in violation of IEEE´s Publication Principles.
We hereby retract the content of this paper. Reasonable effort should be made to remove all past references to this paper.
The presenting author of this paper has the option to appeal this decision by contacting TPII@ieee.org.
Objective: To detect the distribution of ICVD (Ischemic Cerebrovascular Disease) patients and healthy controls MBL haplotype and genotype frequencies, MBL plasma levels. Analyze the relevance between these factors´ change and ICVD and try to explain the possible pathogenesis of ICVD. Methods: MBL haplotypes were detected by SSP-PCR, Plasma MBL levels were detected by ELISA assay. Results: ICVD patients and healthy MBL haplotype and genotype proportions were different (p <;0.05); ICVD plasma MBL levels of patients (3372.18μg / L) was significantly higher (P <;0.01) than in healthy people (2065.29μg / L). Conclusion: MBL may be involved in the development process of ICVD.
Keywords
brain; cardiovascular system; diseases; genetics; ELISA assay; ICVD distribution; ICVD pathogenesis; MBL gene haplotypes; MBL genotype; MBL plasma levels; SSP-PCR; ischemic cerebrovascular disease; Atherosclerosis; Companies; DNA; Diseases; Frequency control; Immune system; Plasmas;
fLanguage
English
Publisher
ieee
Conference_Titel
Bioinformatics and Biomedical Engineering, (iCBBE) 2011 5th International Conference on
Conference_Location
Wuhan
ISSN
2151-7614
Print_ISBN
978-1-4244-5088-6
Type
conf
DOI
10.1109/icbbe.2011.5780424
Filename
5780424
Link To Document